Loss of bombesin-induced feeding suppression in gastrin-releasing peptide receptor-deficient mice.

نویسندگان

  • L L Hampton
  • E E Ladenheim
  • M Akeson
  • J M Way
  • H C Weber
  • V E Sutliff
  • R T Jensen
  • L J Wine
  • H Arnheiter
  • J F Battey
چکیده

The gastrin-releasing peptide receptor (GRP-R) is one of three members of the mammalian bombesin subfamily of seven-transmembrane G protein-coupled receptors that mediate diverse biological responses including secretion, neuromodulation, chemotaxis, and growth. The X chromosome-linked GRP-R gene is expressed widely during embryonic development and predominantly in gastrointestinal, neuronal, and neuroendocrine systems in the adult. Surprisingly, gene-targeted mice lacking a functional GRP-R gene develop and reproduce normally and show no gross phenotypic abnormalities. However, peripheral administration of bombesin at dosages up to 32 nmol/kg to such mice had no effect on the suppression of glucose intake, whereas normal mice showed a dose-dependent suppression of glucose intake. These data suggest that selective agonists for the GRP-R may be useful in inducing satiety.

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عنوان ژورنال:
  • Proceedings of the National Academy of Sciences of the United States of America

دوره 95 6  شماره 

صفحات  -

تاریخ انتشار 1998